61.08 Defective Homologous Recombination in Platinum-based Chemotherapy for Gastric Cancer

T. Katada1, H. Ichikawa1, Y. Hirose1, M. Nagahashi1, Y. Shimada1, T. Hanyu1, T. Ishikawa1, Y. Kano1, Y. Muneoka1, J. Sakata1, T. Kobayashi1, H. Kameyama1, T. Wakai1  1Niigata University Graduate School of Medical and Dental Sciences,Division Of Digestive And General Surgery,Niigata, NIIGATA, Japan

Introduction: Defective homologous recombination (HR) due to genetic alteration of HR genes contributes to a high response of platinum therapy in ovarian and breast cancers. The aim of this study is to clarify the clinical significance of defective HR in platinum-based chemotherapy for gastric cancer (GC).

Methods: A total of 26 patients (19 men and 7 women, with a median age of 66 years) who underwent platinum-based chemotherapy for unresectable metastatic GC were enrolled. Metastatic sites after gastrectomy were lymph node in 10 (38%), liver in 8 (31%), peritoneum in 8 (31%), others in 2 (8%) patients. Genetic alterations of HR genes (BRCA1, BRCA2, RAD50, RAD51C, RAD51D, BLM, PALB2 and FANCD2) in the primary tumor were assessed by cancer gene-panel. We evaluated the association between defective HR and treatment response (RECIST ver. 1.1), progression free survival (PFS) and overall survival (OS).

Results: BRCA1, BRCA2, RAD50, BLM, and FANCD2 mutations were shown in 3 (12%), 2 (8%), 2 (8%), 1 (4%) and 1 patient (4%), respectively. Overall, 6 patients (23%) had genetic alterations of HR genes (defective HR group). Objective response rate (complete or partial response) was 60% in the defective HR group and 27% in the proficient HR group (P = 0.29). Disease control rate (complete or partial response or stable disease) was 100% in the defective HR group and 47% in the proficient HR group (P = 0.06). PFS was significantly longer in the defective HR group than in the proficient HR group (median 6 months vs. 3 months; P = 0.035). OS was also significantly longer in the defective HR group than in the proficient HR group (median 18 months vs. 8 months; P = 0.025).

Conclusions: Genetic alteration of HR genes was significantly associated with a high response of platinum-based chemotherapy and favorable patient outcome in unresectable metastatic GC. Clinical utility of the assessment of defective HR in GC is worth considering further large-scale validation studies.